Our follow-up study on these Fgf10 hypomorphic mice showed that Fgf10 heterozygous newborn mice exposed to hyperoxia (85% hyperoxia, P0–P8) displayed a BPD-like lung phenotype with increased hypoalveolarization accompanied with decreased number of AT2 but an increased number of ATI. Here, FGF10 is linked to bronchopulmonary dysplasia.