As expected, a tumor-endothelial cell adhesion assay identified that forced expression of VCAM1 in RKO and Caco-2 cells caused a significant increase in the ability of these cells to adhere to HUVECs (Figure 4B), while suppression of VCAM1 impaired the adhesion of LoVo and HCT116 cells (Figure 4B). This evidence concerns the gene VCAM1 and neoplasm.