The results showed genes were mainly enriched in the pathways including “cytokine-cytokine receptor interaction,” “Ras signaling pathway,” and “MAPK signaling pathway” for cervical cancer and the pathways including “cytokine-cytokine receptor interaction,” “chemokine signaling pathway,” and “PI3K-Akt signaling pathway” for endometrial cancer, indicating a possible relationship of these IRGs with tumor-associated development, progression, and invasion. The gene discussed is AKT1; the disease is neoplasm.