From this IRG–TF regulatory network, we can see that in cervical cancer, STAT1 positively regulates low-risk IRGs (PSME2, LTA, and PTPN6), but STAT1 positively regulates high-risk IRGs (OAS1) in endometrial cancer, suggesting that transcription factor STAT1 may play different biological roles in these two types of cancers. This evidence concerns the gene TF and endometrial cancer.