Mice that lack expression of the TLR2 (a cell membrane receptor that recognizes peptidoglycan from Gram-positive bacteria) or the myeloid differentiation primary response 88, MYD88 (intracellular adaptor molecule for TLRs), lack innate immune response and were protected from En. faecalis-induced inflammation, steatosis, and liver injury. This evidence concerns the gene TLR2 and steatosis.