As demonstrated in patients with severe acute respiratory syndrome (SARS) more than 15 years ago, elevated levels of T‐helper cell‐originated proinflammatory cytokines, that is, interferon (IFN)‐γ, IL‐1B, IL‐6, IL‐12, IP‐10, neutrophil‐originated chemokine IL‐8, and MCP‐1, were associated with pulmonary inflammation and extensive lung damage.24 The gene discussed is CCL2; the disease is severe acute respiratory syndrome.