Studies have shown that bone marrow stromal cells (BMSCs) overexpress FGL1 to repair acute liver injury by regulating p-STAT/STAT3 [33], and overexpression of FGL-1 was associated with epithelial intermediate transformation and angiogenesis of LKB1-mutant lung adenocarcinoma cells [34]. This evidence concerns the gene FGL1 and lung adenocarcinoma.