Moreover, the recognition of a number of aging‐related tauopathies (aging‐related tau astrogliopathy; primary age related tauopathy), sometimes in patients without clinical deficits, further supports the theory that on occasion, tau deposition may be a late or secondary phenomenon or may merely reflect processes associated with normal aging and life insults.1, 2. The gene discussed is MAPT; the disease is tauopathy.