CSF2 and allergic rhinitis: A study on human nasal epithelial cells (JME/CF15) treated with IL-13 as an in vitro model of allergic rhinitis showed there is association between miR-16 and inflammatory reactions so that miR-16 upregulation in JME/CF15 cells leads to inhibition of cytokines including granulocyte-macrophage colony-stimulating factor (GM-CSF), eotaxin, IL-1β, IL-6, and IL-10.