Moreover, various EBV genes including latent genes contribute to transformation of human B and some oral epithelial cells and are continually expressed in EBV-associated cancer cells, which links EBV with viral oncogenesis [42–44]; for example, expression of EBNA1, LMP1, -2A genes characterize type II latency and is linked with Hodgkin’s lymphoma and some carcinomas like gastric, nasopharyngeal and breast [22, 26]. Here, PDLIM7 is linked to carcinoma.