The high expression of LIMK1 in exosomes facilitated the proliferation and restrained the apoptosis of HCC cells, and the co‐treatment with miR‐520a‐3p mimic counteracted the effects caused by the addition of Over LIMK1‐Propofol‐Huh7‐exo (Figure 8A and B). This evidence concerns the gene LIMK1 and hepatocellular carcinoma.