The combination of the deletions of the mTOR complex 1 (mTORC1) and 2 (mTORC2) by the conditional knockout of the regulatory associated protein of mammalian target of rapamycin (RAPTOR), which is an essential component of mTORC1, and the rapamycin-insensitive companion of mammalian target of rapamycin (RICTOR), which is essential for mTORC2, resulted in the development of a Fanconi-like syndrome involving glucosuria in mice [57]. Here, MTOR is linked to Glycosuria.