Coincidently, a recent study also found that overexpressed ZFP36 could decrease EMT and colony formation in vitro, and promote chemosensitivity to doxorubicin or sorafenib (Krohler et al., 2019), which further suggested the positive role of ZFP36 in upregulating chemosensitivity of HCC cells. The gene discussed is ZFP36; the disease is hepatocellular carcinoma.