In human NASH patients, hepatic CXCL9, CXCL10, CXCL11, CCL3, CCL4, and CCL5 levels were markedly higher than that of the healthy controls and the level of circulating CXCL10 was correlated with the severity of lobular inflammation (74, 75). The gene discussed is CXCL10; the disease is metabolic dysfunction-associated steatohepatitis.