In the article that our study group published earlier [5], the conclusion reached was that despite that both groups suffered from the syndrome, patients who were diagnosed with periodontitis presented certain degrees of individual susceptibility stemming from the differential expressions of genes such as TNFSF13B, ITGB2, ANXA5 and ANXA3, as well as their involvement in immune and/or inflammatory disorders. Here, ANXA3 is linked to periodontitis.