The notion that parotid MALT B-cell lymphomas of pSS patients also express FcRL4, and that LELs are a characteristic feature of salivary gland MALT lymphomas, support the hypothesis that neoplastic MALT B-cells may arise from glandular FcRL4+ intraductal B-cells (15). This evidence concerns the gene FCRL4 and B-cell non-Hodgkin lymphoma.