ACADVL and Hepatic steatosis: Mice deficient in MCAD and VLCAD, the acyl-CoA dehydrogenases involved in the beta dehydrogenation of the acyl-CoA ester derived from fatty acid in the process of β-oxidation, develop hepatic steatosis even fed by chow diet, emphasizing the role of these proteins and fatty acid β-oxidation in the regulation of hepatic TG content34.