Concerning the SLC2A9 and SLC22A12 genes, we were particularly interested in nonsynonymous variants (p.G25R, p.T275M, p.D281H, p.V282I, p.R294H, p.P350L) and other variants known from the literature to be associated with hyperuricemia and gout, or vice versa, i.e., to reduce the risk of gout, namely p.N82N, p.H86H, p.H142H, p.L108L, p.I168I, c.1002+78A>G, and c.535+67A>G [18,19,20,21,22,23]. This evidence concerns the gene SLC2A9 and hyperuricemia.