SOD1 and liver disorder: In another study, we found that postoperative BA patients and a median age of 5.2 years had significantly increased production of ROS, including higher serum SOD, urine 8-iso-PGF2α, and 8-OHdG, than individuals without BA.[9] However, studies on serum and urinary ROS in the pediatric population with liver disease are few,[9–11] and those measuring serum SOD, urinary 8-iso-PGF2α, and urinary 8-OHdG levels are even more scarce.