Some authors have demonstrated its positive effects on reducing the IL-6 plasma concentration, myeloperoxidase activity (MPO), the creatine phosphokinase (CPK) concentration, and the NF-κB expression level in rats with experimentally induced MI, along with inhibition of the pro-inflammatory cytokine TNF-α level in the serum [156]. The gene discussed is NFKB1; the disease is myocardial infarction.