Accordingly, in DMD patients, post-receptor mechanisms underlying scotopic and photopic vision and ON- and OFF-pathway responses measured by electroretinography (ERG) may be altered (also depending on the site of dys mutation), suggesting that dystrophins are required for normal electrophysiology and for proper function of luminance and red-green cone opponent mechanisms in the human retina [9, 32–37]. The gene discussed is DMD; the disease is Duchenne muscular dystrophy.