HDAC4 and gastric cancer: Lu et al. indicated that lncRNA BC032469 acted as a novel ceRNA to modulate hTERT expression by sponging miR-1207-5p and promoted proliferation in gastric cancer 33; Li et al. found that MIAT competitively binded to miR-29a-3p and consequently upregulated the expression of HDAC4, highlighting the involvement of the MIAT/miR-29a-3p/HDAC4 axis in the cell biological behaviors of GC 34; LincHOTAIR epigenetically repressed miR34a by binding to PRC2 and contributed to the epithelial-to-mesenchymal transition (EMT), finally accelerating metastasis in human gastric cancer 35.