While our metabolomics and transcriptomic results support the phospholipid, mitochondria and glutamate dysfunction hypotheses of schizophrenia, these results point at similar changes occurring in schizophrenia and autism (Serpina3n, Gfap) as evidenced by the linkage analysis of the transcriptomics of the MET mice with the gene expression changes in schizophrenia and autism patients. Here, GFAP is linked to autism.