Nevertheless, GO analyses of the affected genes at a nominal significance threshold unraveled coherent changes reflecting hampered transcriptional and translational processes and enhanced migratory ability in BAL cells of MS patients, the latter being driven by upregulation of several pro-inflammatory genes, MS-associated genes (e.g. osteopontin), and adhesion/migration molecules such as MMPs and Ninjurin 1. Here, NINJ1 is linked to myeloid sarcoma.