Senba28 performed cocultures by direct contact with MKN‐7 cells derived from differentiated gastric adenocarcinoma and UE6E7T‐12 cells derived from BM‐MSCs, and indicated that BM‐MSCs induce EMT of gastric cancer cells and increase the number of CSCs by showing increased expression of EMT transcription factor vimentin, Snail, and CD133 that are markers of CSCs. The gene discussed is VIM; the disease is gastric adenocarcinoma.