Based on the above analysis, the results of this study suggest that the lncRNA AC068234.2–202 is a cis-acting regulator of ITGB3 and the lncRNA AP001033.3–201 is a trans-acting regulator of TBXA2R, contributing to the induction of platelet activation and subsequently to the pathogenesis of CAD. The gene discussed is ITGB3; the disease is coronary artery disorder.