Mutp53 promotes proliferation and growth capacity of CSC-like cells and increases CSC markers (CD133, CD44, and YAP/TAZ) in glioblastoma and breast cancer cells by regulating WASP-interacting protein (WIP), which in turn stabilizes YAP/TAZ (Escoll et al., 2017). This evidence concerns the gene WIPF1 and glioblastoma.