Maintenance of telomeres’ length, required for the unlimited cell proliferation displayed by cancer cells, is provided by telomerase, a ribonucleoprotein complex containing a specialized reverse transcriptase, encoded by the telomerase reverse transcriptase (TERT) gene, which uses an internal RNA template (TR) to maintain telomeres’ length, thus playing a critical role in tumor formation and progression [1]. Here, TERT is linked to neoplasm.