These include: the specific recognition of tumor-associated antigens and the expression of effector molecules by T lymphocytes (type I and II- interferon, perforin, Fas/FasL, tumor necrosis factor (TNF)-related apoptosis-inducing ligand—TRAIL), analogously to a viral infection, paralleled by the expression of recognition molecules such as NKG2D or ligands on tumor cells (induced by DNA damage and stress pathways) (117). Here, FAS is linked to neoplasm.