IL-17A is primarily produced by γδT cells in the infarcted heart [27] and are involved in late remodeling stages after MI by promoting a sustained infiltration of neutrophils and macrophages and upregulation of pro-inflammatory cytokines leading to cardiomyocyte death and fibrosis [28] via the MMP/TIMP signaling pathway [29]. This evidence concerns the gene IL17A and myocardial infarction.