More recently, it was reported that inflationary KLRG1+CD62L− cells in latent infection differ from KLRG1+CD62L− SLEC of the acute response, in that they have an extended life span due to IL-15-mediated expression of the anti-apoptotic protein Bcl-2, which makes them ‘memory cell-like’ [35]. This evidence concerns the gene KLRG1 and disease arising from reactivation of latent virus.