EPO and Hyperglycemia: By diminution of hyperglycaemia and restoration of the tubuloglomerular feedback, energy and oxygen demand and renal metabolic stress are reduced and myofibroblasts are proposed to revert back to erythropoietin‐producing fibroblasts, effectively increasing serum erythropoietin and haemoglobin, augmenting oxygen delivery to renal as well as cardiac cells.67