EPO and Hyperglycemia: A current theory revolves around reduction in metabolic stress in the kidney by SGLT‐2is: metabolic stress (increased energy and oxygen demand) due to hyperglycaemia in DM may cause erythropoietin‐producing fibroblasts near the proximal tubules in the kidney to transform into myofibroblasts which produce fibrogenic molecules, resulting in decreased serum erythropoietin levels, positively correlating with increasing HbA1c.67