A study reported with EOCc and citral produced a decreased expression of Bcl-xL and Mcl-1 antiapoptotic factors, in turn, suppressed the proliferation/survival of small lung cancer cells (SCLC cells), [23,39] also decreased cell proliferation, increasing intracellular (reactive oxygen species) ROS, altering the potential of mitochondrial membrane, and initiating apoptosis in cervical cancer cell lines HeLa and ME-180 [40]. Here, BCL2L1 is linked to cervical carcinoma.