KDM1A and acute myeloid leukemia: The three key targets to inhibit are the function of the epigenetic writers (disruptor of telomeric silencing 1-like histone methyltransferase (DOT1L)), epigenetic erasers (lysine-specific histone demethylase 1 (LSD1)), and epigenetic readers (the bromodomain and extra-terminal motif (BET)) in AML patients.