It has been suggested that CASK may act via its interaction with Tbr1 (Hsueh, Wang, Yang, & Sheng, 2000), but in a murine model, disruption of the CASK‐Tbr1 interaction did not produce any structural defects or epilepsy (Huang & Hsueh, 2016). The gene discussed is CASK; the disease is epilepsy.