To exclude the possibility that HpARI_CCP1/2 is interfering with the Alternaria allergen directly, exacerbating the response to it, we used a second model of IL-33-dependent responses (21–23), infecting mice with Nippostrongylus brasiliensis and administering HpARI or HpARI_CCP1/2 to the lungs during the first 3 days of infection. Here, IL33 is linked to infection.