MYD88 and acute respiratory distress syndrome: LPS challenge led to a significant increase in LPS-induced expressions of TLR, myeloid differentiation factor 88 (MyD88), Cox2 and phospho-inhibitor of κBβ (IκBβ), while narciclasine treatment led to a significant reduction in expressions of TLR, MyD88, Cox2 and p-IκBβ in lung tissue of neonatal ALI rats, with no significant difference in total IκBβ expression.