Cystic fibrosis (CF), an autosomal recessive disorder occurring in 1/2000–3000 livebirths among the Caucasian populations [1], is caused by mutations of the gene encoding for the Cystic Fibrosis Transmembrane Conductance Regulator (CFTR), a cyclic adenosine monophosphate–regulated ion channel, which primarily acts as a chloride channel and controls the movement of salt and water into and out of the epithelial cells of several glands. Here, CFTR is linked to cystic fibrosis.