For this reason, we have performed a study in which we have analysed some of the markers most broadly studied in the context of tuberculosis, such as IFNγ or the IFN-inducible protein 10 (IP-10), but also other cytokines, chemokines and growth factors that could be contributing to the infection, and therefore present a distinct signature on the different study groups. This evidence concerns the gene CXCL10 and tuberculosis.