CTNNB1 and neoplasm: Our anoikis analysis also revealed that MGC 80-3 cells with MYH9 knockdown (MYH9 shRNA3 + vector) had an increased rate of apoptosis when compared with that of the control group (NC + vector), whereas restoration of MYH9 or CTNNB1 expression (MYH9 shRNA3 + MYH9 or MYH9 shRNA3 + CTNNB1) blocked tumor cell apoptosis (Figure 6E, S20C).