ATP4B and neoplasm: Although our transgenic GC mouse models (LSL-hMYH9; Atp4b-cre; Tff1-/- mice and Myh9fl/fl; Atp4b-cre; Tff1-/- mice) supported the finding that MYH9-mediated CTNNB1 transcription promotes tumor progression (proliferation and invasion) and shortens the survival of Tff1-/- mice, we failed to find peritoneal metastasis in our models.