However, apoptosis FACS of nondividing CFSEMax CD34+ cells revealed a significant increase in the quiescent CML cell population (Fig. 5f, Supplementary Figs. S15 + S16), showing that combined treatment not only reduces the stem cell quality of CML cells, but also induces apoptosis in this therapy-resistant cell population. The gene discussed is CD34; the disease is chronic myelogenous leukemia, BCR-ABL1 positive.