Case in point, a study in which the effects of miR-205 were analyzed in brains of sporadic PD patients, cell lines, primary neuron cultures, and mouse midbrain dopaminergic neurons proved that it can drop the expression of the LRRK2 protein owing to a conserved binding site located in the untranslated region of the 3′ extreme of the LRRK2 gene and prevent the defects of the neurite outgrowth [62]. Here, LRRK2 is linked to Parkinson disease.