These findings for an in vitro model suggest that in STEMI and post-STEMI patients, IL-17 induces the recruitment and activation of monocyte subsets through an increase in TLR4 and IL-6 secretion, which might cause damage to myocardial tissue in STEMI and post-STEMI. The gene discussed is IL17A; the disease is ST Elevation Myocardial Infarction.