This sign of inflammatory cell death, considered along with the changes in phosphorylation of JNK1 and caspase-3 and the absence of kinases that regulate cell death inhibitors like IkB-alpha and MNK at 30 min p.i. (Table 4), suggests that S. Enteritidis may cause more cell death than S. Heidelberg during initial infection (Table 5). Here, CASP3 is linked to infection.