While this research was ongoing, a study was published supporting our data by demonstrating in the ERα-negative SKBR3 breast cancer cells that the environmental pollutant 3-methylcholantrene, mainly known to exert its carcinogenic effects through AhR, stimulates cell growth response through a functional interaction between AhR and GPR30 (74). This evidence concerns the gene GPER1 and breast carcinoma.