Consistently, our study revealed an increased MYH6 signal that it is specific to a slow developmental myosin isoform derived from PM muscle (Miller et al., 1985) in both WS and WB muscles (not shown), arguing in favor of a shift from fast- to slow-twitching as already suggested (Papah and Abasht, 2019). The gene discussed is MYH6; the disease is Werner syndrome.