Amazingly, the GSVA suggested that high SNORS groups were enriched in signalling pathways associated with “TGF-β signalling”, “MTORC1 signalling”, “epithelial-mesenchymal transition (EMT)”, and “KRAS signalling up”, etc., which were representative of ECM modification and malignancy of BLCA we have reported previously [58]. Here, KRAS is linked to bladder transitional cell carcinoma.