This additional analysis showed that the strength of the relationships between the key risk factors (namely, γ-GT, ALT, AST, gender, and e-GFR) with the outcome variable (endothelial dysfunction) we found in the whole study population (see Table 4) was of similar magnitude to that found in subgroup A (see Table 5) and subgroup B (see Table 6) of the sensitivity analysis. The gene discussed is GPT; the disease is endothelial dysfunction.