Another novel genomic rearrangement, resulting in the chimeric protein NPM1-HAUS1 (Augmin-Like Complex Subunit 1), has been identified in AML patients [61], while an interstitial deletion of 5q associated with NPM1 haploinsufficiency has been observed in myelodysplastic syndromes (MDS) [62] but also found in AML [63] and in T-cell acute lymphoblastic leukemia (T-ALL) [64]. The gene discussed is NPM1; the disease is T-cell acute lymphoblastic leukemia.