Proposed mechanisms of endothelial dysfunction related to postprandial hyperglycemia and diabetes involve alterations in arginine [11], branched chain amino acid (BCAA) [12], and free fatty acid (FFA) metabolism [13], and changes in gut hormones such as the glucagon-like peptide-1 (GLP-1) [14]. The gene discussed is GCG; the disease is Hyperglycemia.