ACE2 and COVID-19: This is in accordance with a recently published series describing postmortem findings of firm and severely congested lung parenchyma with concomitant exudative diffuse alveolar damage.[9] The selective tropism of the COVID-19 for the angiotensin converting enzyme 2 (ACE2), exposed in type II alveolar cells, results in cells damage and apoptosis with consequent acute alveoli injury, septum disruption, and pulmonary edema.